老年痴呆症 z-bo 2020-10-21 275

　　An international research team composed of 31 Alzheimer's disease experts published an article stating that the current focus of Alzheimer's disease research should be appropriately shifted. Their ideas are clear. Given that our research on the prevention and treatment of Alzheimer's disease has been unsuccessful for decades, it is time to consider the possibility of microbial transmission of Alzheimer's disease.

　　In the article, they emphasized the role of herpes virus, the virus that causes frost infection, and the impact of two bacteria on Alzheimer's disease.

　　This discussion is not new. Previous research has shown that patients with Alzheimer's disease are susceptible to infection by certain microorganisms, including certain fungi.

　　However, so far, no breakthrough has been made in the research on this correlation. It is now generally said that the misfolding of tau protein and the accumulation of amyloid plaques in the brain destroy the connections between neurons, leading to memory loss and cognitive decline, until death. These scientists believe that certain viruses and bacteria will directly cause the accumulation of amyloid plaques, and hope to find specific antibacterial agents to prevent the deterioration of this process. I will. We need to focus on simple herpes viruses (HSV-1), chlamydia and spirochetes. According to the article, there are currently more than 100 studies on the relationship between HSV-1 and Alzheimer's disease.

　　If this hypothesis is confirmed, the company can explain why surgery and blood exchange can spread the disease.

　　So how do viruses and bacteria cause Alzheimer's disease? Still don't know. As a result, the field is currently stagnant. However, it is currently known that HSV-1 can damage the nervous system, and viral infections can also cause systemic inflammation. These are symptoms of Alzheimer's disease.

　　However, many scientists do not understand the relationship between the two, so they choose to start from another angle. Neuroscientist John Hardy said: "This is not a mainstream view." "The discussion in this article does not represent the views of most neuroscientists on Alzheimer's disease."

　　"I wrote this article because I ignored important aspects of the etiology of Alzheimer's disease. In fact, clinical treatment results show that anti-infective treatment can effectively delay the progression of Alzheimer's disease.

　　Of course, the problem is not as simple as explained in the article. If so, we have found a cure for this disease. Most importantly, experts call on the public to warn that "Alzheimer's disease can be spread."

　　Of course, this discussion provides good indicators for future research, but there is no clear evidence that Alzheimer's disease can actually be transmitted through microorganisms," said the director of the Alzheimer's Association in the United Kingdom. James Picker Special (James Pickett) said.