糖尿病 z-bo 2020-08-17 538

　　Previous reports indicate that the innate immune cells of non-obese diabetic mice are defective, and the Toll-like receptor TLR4 plays a role in preventing type I diabetes. Williamidgway, chair of the Department of Immunology, Allergy and Rheumatology at the University of Cincinnati, said his research team used the monoclonal antibody UT18 to enhance TLR4 activity and help people with new diabetes develop diabetes. Said to have overthrown most. Diabetes is newly developed in obese mice.

　　Satokiyin: "Our research shows that using antibodies to stimulate specific molecules in the innate immune system can trigger new diabetes in mice that have already developed diabetic symptoms. It can be reversed and has a high success rate. This reversal is successful. Yes, because the beta cells of the pancreas are maintained during autoimmune attacks. The time from the onset of type I diabetes to the end is still relatively short.